Coxiella burnetii Employs the Dot/Icm Type IV Secretion System to Modulate Host NF-κB/RelA Activation

Saugata Mahapatra; Brandi Gallaher; Sydni Caet Smith; Joseph G. Graham; Daniel E. Voth; Edward I. Shaw

doi:10.3389/fcimb.2016.00188

Coxiella burnetii Employs the Dot/Icm Type IV Secretion System to Modulate Host NF-κB/RelA Activation

Saugata Mahapatra, Brandi Gallaher, Sydni Caet Smith, Joseph G. Graham, Daniel E. Voth, Edward I. Shaw

Department of Bio-Medical Sciences (PCOM)

Research output: Contribution to journal › Article › peer-review

Original language	American English
Journal	Frontiers in Cellular and Infection Microbiology
Volume	6
DOIs	https://doi.org/10.3389/fcimb.2016.00188
State	Published - Dec 19 2016

Disciplines

Virology
Biology
Immunology and Infectious Disease

Access to Document

10.3389/fcimb.2016.00188License: Unspecified

Cite this

@article{86f05f4be6be4effa54343727442d812,

title = "Coxiella burnetii Employs the Dot/Icm Type IV Secretion System to Modulate Host NF-κB/RelA Activation",

author = "Saugata Mahapatra and Brandi Gallaher and Smith, {Sydni Caet} and Graham, {Joseph G.} and Voth, {Daniel E.} and Shaw, {Edward I.}",

note = "Coxiella burnetii is the causative agent of Q fever and an obligate intracellular pathogen in nature that survives and grows in a parasitophorous vacuole (PV) within eukaryotic host cells. C. burnetii promotes intracellular survival by subverting apoptotic and pro-inflammatory signaling pathways that are typically regulated by nuclear transcription factor-B (NF-B).",

year = "2016",

month = dec,

day = "19",

doi = "10.3389/fcimb.2016.00188",

language = "American English",

volume = "6",

journal = "Frontiers in Cellular and Infection Microbiology",

}

TY - JOUR

T1 - Coxiella burnetii Employs the Dot/Icm Type IV Secretion System to Modulate Host NF-κB/RelA Activation

AU - Mahapatra, Saugata

AU - Gallaher, Brandi

AU - Smith, Sydni Caet

AU - Graham, Joseph G.

AU - Voth, Daniel E.

AU - Shaw, Edward I.

N1 - Coxiella burnetii is the causative agent of Q fever and an obligate intracellular pathogen in nature that survives and grows in a parasitophorous vacuole (PV) within eukaryotic host cells. C. burnetii promotes intracellular survival by subverting apoptotic and pro-inflammatory signaling pathways that are typically regulated by nuclear transcription factor-B (NF-B).

PY - 2016/12/19

Y1 - 2016/12/19

UR - https://www.frontiersin.org/articles/10.3389/fcimb.2016.00188/full

U2 - 10.3389/fcimb.2016.00188

DO - 10.3389/fcimb.2016.00188

M3 - Article

VL - 6

JO - Frontiers in Cellular and Infection Microbiology

JF - Frontiers in Cellular and Infection Microbiology

ER -

Coxiella burnetii Employs the Dot/Icm Type IV Secretion System to Modulate Host NF-κB/RelA Activation

Disciplines

Access to Document

Other files and links

Cite this