Genetic Inactivation of Chlamydia trachomatis Inclusion Membrane Protein CT228 Alters MYPT1 Recruitment, Extrusion Production, and Longevity of Infection

Jennifer H. Shaw; Charlotte E. Key; Timothy A. Snider; Prakash Sah; Edward I. Shaw; Derek J. Fisher; Erika I. Lutter

doi:10.3389/fcimb.2018.00415

Genetic Inactivation of Chlamydia trachomatis Inclusion Membrane Protein CT228 Alters MYPT1 Recruitment, Extrusion Production, and Longevity of Infection

Jennifer H. Shaw, Charlotte E. Key, Timothy A. Snider, Prakash Sah, Edward I. Shaw, Derek J. Fisher, Erika I. Lutter

Department of Bio-Medical Sciences (PCOM)

Research output: Contribution to journal › Article › peer-review

Original language	American English
Journal	Frontiers in Cellular and Infection Microbiology
Volume	8
DOIs	https://doi.org/10.3389/fcimb.2018.00415
State	Published - Nov 30 2018

Disciplines

Virology
Cell Biology
Biology

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10.3389/fcimb.2018.00415License: Unspecified

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@article{57ce915417004ad9b6a4a8ba236ddac2,

title = "Genetic Inactivation of Chlamydia trachomatis Inclusion Membrane Protein CT228 Alters MYPT1 Recruitment, Extrusion Production, and Longevity of Infection",

author = "Shaw, {Jennifer H.} and Key, {Charlotte E.} and Snider, {Timothy A.} and Prakash Sah and Shaw, {Edward I.} and Fisher, {Derek J.} and Lutter, {Erika I.}",

note = "Chlamydia trachomatis is an obligate intracellular pathogen with global health and economic impact. Upon infection, C. trachomatis resides within a protective niche, the inclusion, wherein it replicates and usurps host cell machinery and resources. The inclusion membrane is the key host-pathogen interface that governs specific protein-protein interactions to manipulate host signaling pathways.",

year = "2018",

month = nov,

day = "30",

doi = "10.3389/fcimb.2018.00415",

language = "American English",

volume = "8",

journal = "Frontiers in Cellular and Infection Microbiology",

}

TY - JOUR

T1 - Genetic Inactivation of Chlamydia trachomatis Inclusion Membrane Protein CT228 Alters MYPT1 Recruitment, Extrusion Production, and Longevity of Infection

AU - Shaw, Jennifer H.

AU - Key, Charlotte E.

AU - Snider, Timothy A.

AU - Sah, Prakash

AU - Shaw, Edward I.

AU - Fisher, Derek J.

AU - Lutter, Erika I.

N1 - Chlamydia trachomatis is an obligate intracellular pathogen with global health and economic impact. Upon infection, C. trachomatis resides within a protective niche, the inclusion, wherein it replicates and usurps host cell machinery and resources. The inclusion membrane is the key host-pathogen interface that governs specific protein-protein interactions to manipulate host signaling pathways.

PY - 2018/11/30

Y1 - 2018/11/30

UR - https://www.frontiersin.org/articles/10.3389/fcimb.2018.00415/full

U2 - 10.3389/fcimb.2018.00415

DO - 10.3389/fcimb.2018.00415

M3 - Article

VL - 8

JO - Frontiers in Cellular and Infection Microbiology

JF - Frontiers in Cellular and Infection Microbiology

ER -

Genetic Inactivation of Chlamydia trachomatis Inclusion Membrane Protein CT228 Alters MYPT1 Recruitment, Extrusion Production, and Longevity of Infection

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